A fascinating study came out of Switzerland in 2018. () What makes this study intriguing is that it was lead by scientists in the University Health Center of Psychiatry, University of Bern. Specifically the Division of Molecular Psychiatry. Molecular Psychiatry seeks to discover biological systems underlying psychiatric disorders and their treatment.
Here the research group provides the biological element, not the psychiatric element, of food poisonings as related to the vagus nerve. "The gastrointestinal tract is continuously challenged with food antigens, possible pathogens, and cooperative digestive microbiota that present a threat aspect for digestive tract swelling. It is highly innervated by vagal fibers (vagus nerve) that connect the main anxious system with the digestive tract body immune system, making (the) vagus (nerve) a significant part of the neuroendocrine-immune axis.
Bani Chander Roland, at the time of this study was a scientist associated with John Hopkins Medical School and Yale University School of Public Health. He and his colleagues released a paper in the Journal of clinical gastroenterology () where they made an interesting post observation. Here is the intriguing observation.
The association between prolonged small bowel transit time and favorable lactulose breath testing (the test to determine the number of germs in the digestion tract) may be beneficial in assisting clients by targeting therapeutic alternatives for those who are not responding to standard therapy. Remarkably, View Details with favorable lactulose breath screening did not always have generalized intestinal motility suggesting that small bowel transit particularly inclines to the development of SIBO.
So here we have an observation. An interruption in the typical flow and transit of foods triggered the SIBO. Let's go a step even more and continue on with research from Dr. Bani Chander Roland. SIBO is an issue of stuck valves and excess acid In the American Journal of Digestion Diseases () Dr.
In this research study the researchers hypothesized that loss of gastric acid, a postponed digestive transit, and ileocecal valve dysfunction may cause the advancement of SIBO. Explanatory note: The ileocecal valve is a sphincter muscle where the little intestine and large intestine satisfy. When the small intestines are done absorbing foods, it passes the remnant item of digested food materials off to the big intestines at the colon.